Steroid hormones regulate gene expression by

Diabetes results from inadequate levels of insulin. Type I diabetes is characterized by inadequate levels of insulin secretion, often due to a genetic cause. Type II usually develops in adults from both genetic and environmental causes. Loss of response of targets to insulin rather than lack of insulin causes this type of diabetes. Diabetes causes impairment in the functioning of the eyes, circulatory system, nervous system, and failure of the kidneys. Diabetes is the second leading cause of blindness in the US. Treatments involve daily injections of insulin, monitoring of blood glucose levels and a controlled diet.

6. Steroid hormones receptor binds to
a) Hormone response elements in m-RNA
b) Hormone response elements in DNA
c) Hormone response elements in proteins
d) Ribosomes to stimulate translation
e) Cell membrane thereby increases permeability
View Answer Answer: b
Explanation: Hormone response elements are short sequence of DNA where steroid hormone receptors bind. 7. Mode of action of steroid hormones involve
a) Stimulation of DNA replication
b) Stimulation of m-RNA transcription
c) Inhibition of protein synthesis
d) Secondary messenger
View Answer Answer: b
Explanation: Steroid hormones involve in the stimulation of m-RNA transcription, affecting the gene expression. 8. The drug antagonist of estrogen is
a) Tanoxifen
b) Metformin
c) Glucophage
d) Victoza
View Answer Answer: a
Explanation: Tanoxifen is an antagonist of estrogen. 9. The drug used to terminate early pregnancies is
a) RU486
b) Metformin
c) Glucophage
d) Victoza
View Answer Answer: a
Explanation: Metformin, Glucophage and victoza are the drugs to cure diabetes. 10. Plasma membrane protein predicted to have seen 7 transmembrane helices segment binds
a) Progesterone
b) Thyroid stimulating hormone
c) Insulin
d) Follicle stimulating hormone
View Answer Answer: a
Explanation: Progesterone hormone is a steroid hormone, can diffuse through cell membranes. Sanfoundry Global Education & Learning Series – Biochemistry.

Finally, there are instances when a progesterone-based hormone therapy might be used in the treatment of trans men. Progesterone may be used in some instances to help stop menstrual flow if testosterone therapy alone does not adequately stop the cycle after a reasonable period of treatment. A short course of progesterone may also be prescribed to induce a shedding of the uterine lining after testosterone therapy has progressed, in the event that there is any unusual buildup of the endometrium. This may help prevent spot bleeding as well as potentially decreasing the risk of uterine cancer. To learn more specifics about FTM testosterone therapy, please read the sections " FTM Testosterone Therapy Basics " and " FTM Testosterone Therapy and General Health ."

Cells of the zona fasciculata and zona reticularis lack aldosterone synthase (CYP11B2) that converts corticosterone to aldosterone, and thus these tissues produce only the weak mineralocorticoid corticosterone. However, both these zones do contain the CYP17A1 missing in zona glomerulosa and thus produce the major glucocorticoid, cortisol. Zona fasciculata and zona reticularis cells also contain CYP17A1, whose 17,20-lyase activity is responsible for producing the androgens, dehydroepiandrosterone (DHEA) and androstenedione. Thus, fasciculata and reticularis cells can make corticosteroids and the adrenal androgens, but not aldosterone.

Steroid hormones regulate gene expression by

steroid hormones regulate gene expression by

Cells of the zona fasciculata and zona reticularis lack aldosterone synthase (CYP11B2) that converts corticosterone to aldosterone, and thus these tissues produce only the weak mineralocorticoid corticosterone. However, both these zones do contain the CYP17A1 missing in zona glomerulosa and thus produce the major glucocorticoid, cortisol. Zona fasciculata and zona reticularis cells also contain CYP17A1, whose 17,20-lyase activity is responsible for producing the androgens, dehydroepiandrosterone (DHEA) and androstenedione. Thus, fasciculata and reticularis cells can make corticosteroids and the adrenal androgens, but not aldosterone.

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